@phdthesis{oai:doshisha.repo.nii.ac.jp:00028496,
 author = {Alam, Shahnur Md},
 month = {2021-10-04},
 note = {Aβ is still best known as a molecule to cause AD through accumulation and deposition within the frontal cortex and hippocampus in the brain that depends on rate of synthesis and clearance of Aβ. Aβ clearance or degradation rather than its synthesis have been found to be more critical in accumulation of Aβ. Therefore, Aβ clearance pathways have been emerged as a new therapeutic target for AD treatment. In the present study indicate that diffusion mediated clearance and drainage from CBL is responsible for less Aβ in CBL than that of CTX. Application of this defense mechanism to CTX would prevent from AD. Although it is not fully clear Aβ about clearance, this finding will draw attention of AD scientist to find out the best possible mechanism after more investigation. Once it may possible, it would be applicable for not only AD pathogenesis but also prevention and cure of AD., application/pdf},
 title = {A potential defense mechanism against amyloid deposition in cerebellum},
 year = {}
}